You are using a browser version with limited support for CSS. The labyrinth is the crucial site of fetal-maternal exchange in the mouse placenta and analogous to the human chorionic villous However, LIF production and localization was not investigated in those studies, so the mechanisms of action in these specific cells could not be determined 32 Indeed in LIFR-mutant fetuses, skeletal and neuronal defects were evident during embryogenesis The area of cytokeratin-positive staining in the placenta, including the junctional zone and labyrinth, was quantified. Salamonsen, L. MMP9-null mice exhibit impaired trophoblast invasion 36suggesting that this protease promotes invasion. Reproduction, fertility and development 21, —, Here we show that LIF inhibition reduced total placental area and also labyrinth area.
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Development and function of trophoblast giant cells in the rodent placenta. To address this hypothesis, we determined the effect of transient LIF blockade during placental formation in mice and the effects on pregnancy outcome.
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Preparing fertile soil: the importance of endometrial receptivity. Cite this article Winship, A. Leukemia inhibitory factor enhances endometrial stromal cell decidualization in humans and mice. We have uncovered the possible mechanisms by which LIF regulates placentation in vivo in mice.
Targeted disruption of the low-affinity leukemia inhibitory factor receptor gene causes placental, skeletal, neural and metabolic defects and results in perinatal death.
(Submitted on 29 Oct ) This demonstrates that biologically-plausible spiking LIF neurons can be integrated into deep networks can perform as well as. VolumeJulyPages. In order to validate the model developed, some selected LiF–CeF3–ThF4 ternary compositions have been synthesized.
Leukemia inhibitory factor triggers activation of signal transducer and activator of transcription 3, proliferation, invasiveness and altered protease expression in choriocarcinoma cells. Similarly, in women the high levels of LIF expression in first trimester decidua and the localization of its receptor on trophoblast cells suggests paracrine actions during placentation 20 Prakash, G.
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Placenta 24, S99— About this article. A Nature Research Journal. Differentiation; research in biological diversity 74, —,
pregnancy loss model, administration of recombinant LIF, combined with.
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 Spiking Deep Networks with LIF Neurons
There were no differences in the number of apoptotic cells at E17 between treatment groups, likely due to the increase in apoptosis in control implantation sites that occurs naturally to facilitate parturition. Gynecological endocrinology: the official journal of the International Society of Gynecological Endocrinology 28, —, Chorioallantoic morphogenesis and formation of the placental villous tree.
There were no differences in placental trophoblast area at E Electronic supplementary material. Kuwabara, M.
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|In accordance, we also observed morphological alterations in these same cell types following LIF inhibition.
J Reprod Immunol 57, — Due to the increased number of resorbed embryos, the maternal blood supply is most likely directed to a smaller number of implantation sites and in rodents, utero-placental blood flow is strongly correlated with fetal weight Kubota, Y.
Interestingly, the surviving fetuses were healthy at birth and until weaning and weighed more than control pups.